Acute inflammatory edema
The proposed pathogenesis of AIE is that acute volume overload in the setting of impaired lymphatic drainage leads to the accumulation of dermal edema. This, in turn, is thought to cause microtrauma to the connective tissue in affected areas, with subsequent influx of inflammatory mediators.
AIE differs from cellulitis in that it is nontender.
R60.9 – Edema, unspecified
103619005 – Inflammatory edema
- Cellulitis – Unilateral, warm, tender plaque in a patient with or without systemic signs of infection.
- Necrotizing fasciitis
- Toxin-mediated erythema, such as staphylococcal scalded skin syndrome or streptococcal toxic shock syndrome
- Exanthematous drug reaction
- Drug reaction with eosinophilia and systemic symptoms (DRESS)
- Viral exanthem
- Symmetric drug-related intertriginous and flexural exanthema (SDRIFE)
- Vancomycin infusion reaction
- Acute graft-versus-host disease
- Cutaneous eruption of lymphocyte recovery
- Engraftment syndrome
- Chronic lymphedema – Chronic lymphedema of the abdomen, as seen accompanying obesity, may present with mild erythema and a peau d'orange appearance.